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In conclusion, this review suggests that the simplistic notion that MDD is caused by reduced GABA neurotransmission must be discarded in favor of a more nuanced and complex model of the role of inhibitory neurotransmission in MDD

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We, therefore, aimed to systematically review the molecular and pathophysiological mechanisms of hyperprolactinemia and obesity, focusing on key central and peripheral pathways, supported by current experimental and clinical evidence ( Figure 1 )